Journal of applied physiology (Bethesda, Md. : 1985)AnimalsMaleRatsSprague-DawleySirolimus
Implications for myofibrillar protein translation due to high-intensity muscle contraction via a rapamycin-insensitive mechanism.
Taiga Mishima, Yuki Fujita, Tomoki Abe, Satoru Ato, Riki Ogasawara
Published: 202610.1152/japplphysiol.00179.2025
Abstract
Muscle protein metabolism is thought to regulate muscle mass. High-intensity muscle contraction (HiMC) increases muscle protein synthesis (MPS), resulting in muscle hypertrophy. Inhibiting the mechanistic target of rapamycin complex 1 (mTORC1) using…
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