m6A epitranscriptomic regulation of KRAS by METTL3 promotes EMT and stromal remodeling through TGF-β/SMAD signaling in cervical cancer.

KRAS N6-methyladenosine (m6A) modification has emerged as a crucial epigenetic regulator in cancer progression, but its role in cervical cancer epithelial-mesenchymal transition (EMT) and stromal remodeling remains unclear. This study explored how ME…

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