Journal of molecular and cellular cardiologyAnimalsMitochondrial Permeability Transition PoreMyocardial Reperfusion InjuryMyocytesCardiac
Nynrin enhances cardiac function by inhibiting mitochondrial permeability transition pore opening upon myocardial ischemia/reperfusion injury.
Yuhan Wang, Yujing Li, Yanan Zhou, Hao Zhang, Jingyi Zang, Chaofan Yang, Zeyu Gao, Yu Hou, Moshi Song
Published: 202510.1016/j.yjmcc.2025.10.002
Abstract
Acute myocardial infarction (AMI) is a leading cause of cardiovascular disease-related death. Reperfusion therapies, although essential, can exacerbate damage through myocardial ischemia/reperfusion (I/R) injury. Cyclophilin D (CypD) and mitochondria…
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