Polycyclic aromatic hydrocarbons-enriched diesel exhaust particles induced hypertrophy in rat primary neonatal cardiomyocytes and apoptotic cell death in H9c2 cardiomyocytes.

There is limited research about the direct effects of diesel exhaust particles (DEPs) on cardiomyocytes. The respirable DEPs asorbed many organic substances such as polycyclic aromatic hydrocarbons (PAHs). Here, we used the standard DEPs, enriched in…

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