Life sciencesAnimalsMuscular DystrophyDuchenneFibrosisMice
HDAC11 deficiency improves muscle phenotype in a Duchenne muscular dystrophy murine model by reducing inflammation and fibrosis.
Renato Odria, Alexandra Mercado-Amarilla, Carolina Soler-Botija, Daniel M Borràs, Jessica Ohana, Pau Maestre-Mora, Anne Bigot, Xavier Suárez-Calvet, Eduard Gallardo, Gisela Nogales-Gadea, Mònica Suelves
Published: 202610.1016/j.lfs.2025.124150
Abstract
Duchenne muscular dystrophy (DMD) is a devastating genetic disorder caused by mutations in the dystrophin gene, which encodes for an essential protein to maintain muscle integrity. Loss of dystrophin leads to progressive muscle degeneration, chronic…
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