European journal of pharmacologyAnimalsFerroptosisSepsisMiceCardiomyopathies
Deficiency of zinc finger protein 36 exacerbates sepsis-induced cardiomyopathy by activating ferroptosis.
Xiaowei Yang, Guangduo Zhu, Ming Li, Haibo Zhan, Feng Wang
Published: 202610.1016/j.ejphar.2025.178405
Abstract
Ferroptosis is an important pathological factor in sepsis-induced cardiomyopathy (SIC). Zinc finger protein 36 (ZFP36) is an RNA-binding protein regulating cellular biological processes by degrading target genes. Studies have shown that ZFP36 is high…
Preview only. Read the full abstract at the source